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Though derived from uveal melanocytes, iris melanomas share more in common with cutaneous (skin) melanomas in that they frequently harbor BRAF mutations associated with ultraviolet damage.

The disease is caused by an increase of melanocytes in the iris, choroid, and surrounding structures.

Transport in Sherbrooke Lentigo maligna is where melanocyte cells have become malignant and grow continuously along the stratum basale of the skin, but have not invaded below the epidermis.

Tumors arise from the pigment cells (melanocytes) that reside within the uvea and give color to the eye.

HMB-45 does not react well against intradermal nevi, normal adult melanocytes, spindle cell melanomas and desmoplastic melanomas.

These diseases often involve a disorder of non-cancerous skin cells, melanocytes, which produce pigment appearing as moles, birthmarks or growths on the surface of the skin or just beneath, which can be rough, flat or raised.

Histopathological examination that shows intraepithelial proliferation of conjunctival epithelial melanocytes.

OCA is caused by mutations in several genes that control the synthesis of melanin within the melanocytes.

There are in addition populations of cells producing somatostatin, angiotensin II and melanocyte stimulating hormone.

It is due to excess production of melanin in the setting of a normal number of melanocytes in the conjunctiva.

The antibody also reacts positively against junctional nevus cells and fetal melanocytes.

These melanocytes are distinct from the retinal pigment epithelium cells underlying the retina that do not form melanomas.

jpg|Light microscopy of lentigo maligna showing the characteristic atypical epidermal melanocytes.

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